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Showing 1 to 12 of 1494 entries
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The synthesis, secretion and uptake of prorenin in human amnion.

Physiological reports

Pringle KG, Wang Y, Lumbers ER.
PMID: 25902786
Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12313.

Very high concentrations of prorenin protein occur in human amniotic fluid and amnion. The source of amniotic fluid prorenin is likely the decidua, as it has the highest levels of prorenin mRNA (REN). Conversely, REN mRNA levels in amnion...

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Pringle KG, Wang Y, Lumbers ER. The synthesis, secretion and uptake of prorenin in human amnion. Physiol Rep. 2015;3(4)doi: 10.14814/phy2.12313.
Pringle, K. G., Wang, Y., & Lumbers, E. R. (2015). The synthesis, secretion and uptake of prorenin in human amnion. Physiological reports, 3(4), . https://doi.org/10.14814/phy2.12313
Pringle, Kirsty G, et al. "The synthesis, secretion and uptake of prorenin in human amnion." Physiological reports vol. 3,4 (2015). doi: https://doi.org/10.14814/phy2.12313
Pringle KG, Wang Y, Lumbers ER. The synthesis, secretion and uptake of prorenin in human amnion. Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12313. PMID: 25902786; PMCID: PMC4425950.
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Effects of decreased lactate accumulation after dichloroacetate administration on exercise training-induced mitochondrial adaptations in mouse skeletal muscle.

Physiological reports

Hoshino D, Tamura Y, Masuda H, Matsunaga Y, Hatta H.
PMID: 26416973
Physiol Rep. 2015 Sep;3(9). doi: 10.14814/phy2.12555.

Recent studies suggested that lactate accumulation can be a signal for mitochondrial biogenesis in skeletal muscle. We investigated whether reductions in lactate concentrations in response to dichloroacetate (DCA), an activator of pyruvate dehydrogenase, attenuate mitochondrial adaptations after exercise training...

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Hoshino D, Tamura Y, Masuda H, et al. Effects of decreased lactate accumulation after dichloroacetate administration on exercise training-induced mitochondrial adaptations in mouse skeletal muscle. Physiol Rep. 2015;3(9)doi: 10.14814/phy2.12555.
Hoshino, D., Tamura, Y., Masuda, H., Matsunaga, Y., & Hatta, H. (2015). Effects of decreased lactate accumulation after dichloroacetate administration on exercise training-induced mitochondrial adaptations in mouse skeletal muscle. Physiological reports, 3(9), . https://doi.org/10.14814/phy2.12555
Hoshino, Daisuke, et al. "Effects of decreased lactate accumulation after dichloroacetate administration on exercise training-induced mitochondrial adaptations in mouse skeletal muscle." Physiological reports vol. 3,9 (2015). doi: https://doi.org/10.14814/phy2.12555
Hoshino D, Tamura Y, Masuda H, Matsunaga Y, Hatta H. Effects of decreased lactate accumulation after dichloroacetate administration on exercise training-induced mitochondrial adaptations in mouse skeletal muscle. Physiol Rep. 2015 Sep;3(9). doi: 10.14814/phy2.12555. PMID: 26416973; PMCID: PMC4600395.
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Cocaine enhances HIV-1 gp120-induced lymphatic endothelial dysfunction in the lung.

Physiological reports

Zhang X, Jiang S, Yu J, Kuzontkoski PM, Groopman JE.
PMID: 26311830
Physiol Rep. 2015 Aug;3(8). doi: 10.14814/phy2.12482.

Pulmonary complications are common in both AIDS patients and cocaine users. We addressed the cellular and molecular mechanisms by which HIV and cocaine may partner to induce their deleterious effects. Using primary lung lymphatic endothelial cells (L-LECs), we examined...

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Zhang X, Jiang S, Yu J, et al. Cocaine enhances HIV-1 gp120-induced lymphatic endothelial dysfunction in the lung. Physiol Rep. 2015;3(8)doi: 10.14814/phy2.12482.
Zhang, X., Jiang, S., Yu, J., Kuzontkoski, P. M., & Groopman, J. E. (2015). Cocaine enhances HIV-1 gp120-induced lymphatic endothelial dysfunction in the lung. Physiological reports, 3(8), . https://doi.org/10.14814/phy2.12482
Zhang, Xuefeng, et al. "Cocaine enhances HIV-1 gp120-induced lymphatic endothelial dysfunction in the lung." Physiological reports vol. 3,8 (2015). doi: https://doi.org/10.14814/phy2.12482
Zhang X, Jiang S, Yu J, Kuzontkoski PM, Groopman JE. Cocaine enhances HIV-1 gp120-induced lymphatic endothelial dysfunction in the lung. Physiol Rep. 2015 Aug;3(8). doi: 10.14814/phy2.12482. PMID: 26311830; PMCID: PMC4562568.
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What is the effect of ischemic preconditioning on the kinetics of pulmonary oxygen uptake and muscle deoxygenation during exercise?.

Physiological reports

Sabino-Carvalho JL, Barbosa TC, Silva BM.
PMID: 26341997
Physiol Rep. 2015 Sep;3(9). doi: 10.14814/phy2.12540.

No abstract available.

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Sabino-Carvalho JL, Barbosa TC, Silva BM. What is the effect of ischemic preconditioning on the kinetics of pulmonary oxygen uptake and muscle deoxygenation during exercise?. Physiol Rep. 2015;3(9)doi: 10.14814/phy2.12540.
Sabino-Carvalho, J. L., Barbosa, T. C., & Silva, B. M. (2015). What is the effect of ischemic preconditioning on the kinetics of pulmonary oxygen uptake and muscle deoxygenation during exercise?. Physiological reports, 3(9), . https://doi.org/10.14814/phy2.12540
Sabino-Carvalho, Jeann L C, et al. "What is the effect of ischemic preconditioning on the kinetics of pulmonary oxygen uptake and muscle deoxygenation during exercise?." Physiological reports vol. 3,9 (2015). doi: https://doi.org/10.14814/phy2.12540
Sabino-Carvalho JL, Barbosa TC, Silva BM. What is the effect of ischemic preconditioning on the kinetics of pulmonary oxygen uptake and muscle deoxygenation during exercise?. Physiol Rep. 2015 Sep;3(9). doi: 10.14814/phy2.12540. PMID: 26341997; PMCID: PMC4600384.
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Reply to the letter from Dr. Miao et al.

Physiological reports

Tanimura Y, Aoi W, Takanami Y, Kawai Y, Mizushima K, Naito Y, Yoshikawa T.
PMID: 27634109
Physiol Rep. 2016 Sep;4(17). doi: 10.14814/phy2.12964.

No abstract available.

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Tanimura Y, Aoi W, Takanami Y, et al. Reply to the letter from Dr. Miao et al. Physiol Rep. 2016;4(17)doi: 10.14814/phy2.12964.
Tanimura, Y., Aoi, W., Takanami, Y., Kawai, Y., Mizushima, K., Naito, Y., & Yoshikawa, T. (2016). Reply to the letter from Dr. Miao et al. Physiological reports, 4(17), . https://doi.org/10.14814/phy2.12964
Tanimura, Yuko, et al. "Reply to the letter from Dr. Miao et al." Physiological reports vol. 4,17 (2016). doi: https://doi.org/10.14814/phy2.12964
Tanimura Y, Aoi W, Takanami Y, Kawai Y, Mizushima K, Naito Y, Yoshikawa T. Reply to the letter from Dr. Miao et al. Physiol Rep. 2016 Sep;4(17). doi: 10.14814/phy2.12964. PMID: 27634109; PMCID: PMC5027365.
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Unexpected effects of the MIP-CreER transgene and tamoxifen on β-cell growth in C57Bl6/J male mice.

Physiological reports

Carboneau BA, Le TD, Dunn JC, Gannon M.
PMID: 27670405
Physiol Rep. 2016 Sep;4(18). doi: 10.14814/phy2.12863.

Transgenic mouse models have been fundamental in the discovery of factors that regulate β-cell development, mass, and function. Several groups have recently shown that some of these models display previously uncharacterized phenotypes due to the transgenic system itself. These...

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Carboneau BA, Le TD, Dunn JC, et al. Unexpected effects of the MIP-CreER transgene and tamoxifen on β-cell growth in C57Bl6/J male mice. Physiol Rep. 2016;4(18)doi: 10.14814/phy2.12863.
Carboneau, B. A., Le, T. D., Dunn, J. C., & Gannon, M. (2016). Unexpected effects of the MIP-CreER transgene and tamoxifen on β-cell growth in C57Bl6/J male mice. Physiological reports, 4(18), . https://doi.org/10.14814/phy2.12863
Carboneau, Bethany A, et al. "Unexpected effects of the MIP-CreER transgene and tamoxifen on β-cell growth in C57Bl6/J male mice." Physiological reports vol. 4,18 (2016). doi: https://doi.org/10.14814/phy2.12863
Carboneau BA, Le TD, Dunn JC, Gannon M. Unexpected effects of the MIP-CreER transgene and tamoxifen on β-cell growth in C57Bl6/J male mice. Physiol Rep. 2016 Sep;4(18). doi: 10.14814/phy2.12863. PMID: 27670405; PMCID: PMC5037909.
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The most sensitive inputs to cutaneous representing regions of primary somatosensory cortex do not change with behavioral training.

Physiological reports

Blake DT, Spingath E.
PMID: 26634900
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12623.

Learning a sensory detection task leads to an increased primary sensory cortex response to the detected stimulus, while learning a sensory discrimination task additionally leads to a decreased sensory cortex response to the distractor stimulus. Neural responses are scaled...

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Blake DT, Spingath E. The most sensitive inputs to cutaneous representing regions of primary somatosensory cortex do not change with behavioral training. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12623.
Blake, D. T., & Spingath, E. (2015). The most sensitive inputs to cutaneous representing regions of primary somatosensory cortex do not change with behavioral training. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12623
Blake, David T, and Spingath, Elsie. "The most sensitive inputs to cutaneous representing regions of primary somatosensory cortex do not change with behavioral training." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12623
Blake DT, Spingath E. The most sensitive inputs to cutaneous representing regions of primary somatosensory cortex do not change with behavioral training. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12623. PMID: 26634900; PMCID: PMC4760438.
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Ascorbic acid attenuates the pressor response to voluntary apnea in postmenopausal women.

Physiological reports

Randolph BJ, Patel HM, Muller MD.
PMID: 25907792
Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12384.

We recently demonstrated that postmenopausal women have an augmented blood pressure response to voluntary apnea compared to premenopausal women. Both obstructive sleep apnea (OSA) and healthy aging are associated with increased oxidative stress, which may impair cardiovascular function. Restoring...

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Randolph BJ, Patel HM, Muller MD. Ascorbic acid attenuates the pressor response to voluntary apnea in postmenopausal women. Physiol Rep. 2015;3(4)doi: 10.14814/phy2.12384.
Randolph, B. J., Patel, H. M., & Muller, M. D. (2015). Ascorbic acid attenuates the pressor response to voluntary apnea in postmenopausal women. Physiological reports, 3(4), . https://doi.org/10.14814/phy2.12384
Randolph, Brittney J, et al. "Ascorbic acid attenuates the pressor response to voluntary apnea in postmenopausal women." Physiological reports vol. 3,4 (2015). doi: https://doi.org/10.14814/phy2.12384
Randolph BJ, Patel HM, Muller MD. Ascorbic acid attenuates the pressor response to voluntary apnea in postmenopausal women. Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12384. PMID: 25907792; PMCID: PMC4425983.
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Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E.

Physiological reports

Díaz V, Peinado AB, Barba-Moreno L, Altamura S, Butragueño J, González-Gross M, Alteheld B, Stehle P, Zapico AG, Muckenthaler MU, Gassmann M.
PMID: 26243212
Physiol Rep. 2015 Aug;3(8). doi: 10.14814/phy2.12475.

Iron deficiency among endurance athletes is of major concern for coaches, physicians, and nutritionists. Recently, it has been observed that hepcidin, the master regulator of iron metabolism, was upregulated after exercise and was found to be related to interleukin-6...

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Díaz V, Peinado AB, Barba-Moreno L, et al. Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E. Physiol Rep. 2015;3(8)doi: 10.14814/phy2.12475.
Díaz, V., Peinado, A. B., Barba-Moreno, L., Altamura, S., Butragueño, J., González-Gross, M., Alteheld, B., Stehle, P., Zapico, A. G., Muckenthaler, M. U., & Gassmann, M. (2015). Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E. Physiological reports, 3(8), . https://doi.org/10.14814/phy2.12475
Díaz, Víctor, et al. "Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E." Physiological reports vol. 3,8 (2015). doi: https://doi.org/10.14814/phy2.12475
Díaz V, Peinado AB, Barba-Moreno L, Altamura S, Butragueño J, González-Gross M, Alteheld B, Stehle P, Zapico AG, Muckenthaler MU, Gassmann M. Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E. Physiol Rep. 2015 Aug;3(8). doi: 10.14814/phy2.12475. PMID: 26243212; PMCID: PMC4562561.
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17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice.

Physiological reports

Yatomi M, Hisada T, Ishizuka T, Koga Y, Ono A, Kamide Y, Seki K, Aoki-Saito H, Tsurumaki H, Sunaga N, Kaira K, Dobashi K, Yamada M, Okajima F.
PMID: 26660549
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12628.

Idiopathic pulmonary fibrosis (IPF) is a destructive inflammatory disease with limited therapeutic options. Inflammation plays an integral role in the development of pulmonary fibrosis. Unresolved inflammatory responses can lead to substantial tissue injury, chronic inflammation, and fibrosis. The resolvins...

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Yatomi M, Hisada T, Ishizuka T, et al. 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12628.
Yatomi, M., Hisada, T., Ishizuka, T., Koga, Y., Ono, A., Kamide, Y., Seki, K., Aoki-Saito, H., Tsurumaki, H., Sunaga, N., Kaira, K., Dobashi, K., Yamada, M., & Okajima, F. (2015). 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12628
Yatomi, Masakiyo, et al. "17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12628
Yatomi M, Hisada T, Ishizuka T, Koga Y, Ono A, Kamide Y, Seki K, Aoki-Saito H, Tsurumaki H, Sunaga N, Kaira K, Dobashi K, Yamada M, Okajima F. 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12628. PMID: 26660549; PMCID: PMC4760456.
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Blunted hypothalamic ghrelin signaling reduces diet intake in rats fed a low-protein diet in late pregnancy.

Physiological reports

Gao H, Sisley S, Yallampalli C.
PMID: 26660550
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12629.

Diet intake in pregnant rats fed a low-protein (LP) diet was significantly reduced during late pregnancy despite elevated plasma levels of ghrelin. In this study, we hypothesized that ghrelin signaling in the hypothalamus is blunted under a low-protein diet...

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Gao H, Sisley S, Yallampalli C. Blunted hypothalamic ghrelin signaling reduces diet intake in rats fed a low-protein diet in late pregnancy. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12629.
Gao, H., Sisley, S., & Yallampalli, C. (2015). Blunted hypothalamic ghrelin signaling reduces diet intake in rats fed a low-protein diet in late pregnancy. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12629
Gao, Haijun, et al. "Blunted hypothalamic ghrelin signaling reduces diet intake in rats fed a low-protein diet in late pregnancy." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12629
Gao H, Sisley S, Yallampalli C. Blunted hypothalamic ghrelin signaling reduces diet intake in rats fed a low-protein diet in late pregnancy. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12629. PMID: 26660550; PMCID: PMC4760445.
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Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity.

Physiological reports

Ali MI, Chen X, Didion SP.
PMID: 26660551
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12630.

Heterozygous endothelial nitric oxide synthase (eNOS) deficiency is associated with normal endothelium-dependent responses, however, little is known regarding the mechanisms that maintain or impair endothelial function with heterozygous eNOS deficiency. The goals of this study were to (1) determine...

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Ali MI, Chen X, Didion SP. Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12630.
Ali, M. I., Chen, X., & Didion, S. P. (2015). Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12630
Ali, M Irfan, et al. "Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12630
Ali MI, Chen X, Didion SP. Heterozygous eNOS deficiency is associated with oxidative stress and endothelial dysfunction in diet-induced obesity. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12630. PMID: 26660551; PMCID: PMC4760452.
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Showing 1 to 12 of 1494 entries